Atlas of Liver Pathology by Anthony W. H. Chan, Alberto Quaglia, Beate Haugk, Alastair PDF

By Anthony W. H. Chan, Alberto Quaglia, Beate Haugk, Alastair Burt

ISBN-10: 1461491134

ISBN-13: 9781461491132

The liver is a posh organ because of its distinctive microscopic constitution, complicated metabolic features and susceptibility to a wide selection of insults, manifesting in numerous histological  patterns.  Atlas of Liver Pathology considers either adjustments obvious in clinical liver biopsies in addition to lesional biopsies whilst the specimen has been taken from a mass. The ebook starts off by way of reviewing common constitution and its versions and the optimum methods for the training of histological sections for diagnostic liver pathology. the subsequent chapters are devoted to developmental, metabolic, infectious, drug similar, autoimmune, biliary, vascular and neoplastic issues. sections on liver pathology in being pregnant and transplantation finish the paintings. Macroscopic illustrations are integrated the place applicable. All pictures are complemented through legends describing the image and delivering appropriate comparable information.

Authored by way of nationally and the world over well-known pathologists, Atlas of Liver Pathology is a priceless source that serves as a short reference advisor for the prognosis of ordinary and strange diseases.

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Bile duct epithelial cells begin to express another biliary cytokeratin (CK7) from the 20th week of gestation, whereas uninvolved hepatocytes in the ductal plate lose CK19. Fig. 4 Extramedullary haematopoiesis in a fetal liver. Extensive sinusoidal extramedullary haematopoiesis is present among a twin-cell plate of hepatocytes. Extramedullary haematopoiesis is a normal physiologic finding during fetal development and normally ceases within a month after birth. Erythropoiesis tends to occur along the hepatic sinusoids, whereas leucopoiesis and thrombopoiesis are found more commonly in portal tracts.

Choledochal cysts can contain a smooth muscle layer allowing distinction from the inflamed, dilated common bile duct observed in obstruction. Premalignant lesions may occur and progress to invasive malignancy, most commonly cholangiocarcinoma, less frequently anaplastic carcinoma and squamous cell carcinoma. 3 Developmental Abnormalities Paucity of Intrahepatic Bile Ducts and Biliary Atresia Fig. 24 Nonsyndromic paucity of intrahepatic bile ducts. This portal tract is devoid of an interlobular bile duct (ductopaenia).

The liver is enlarged with multiple nodules separated by thick fibrous bands. No cystic dilatation of bile ducts is noted grossly. Congenital hepatic fibrosis is mostly the hepatic presentation of a multisystem disorder, and rarely presents in isolation. Juvenile or adult presentation of autosomal recessive polycystic kidney disease accounts for majority of congenital hepatic fibrosis. g. g. Jeune syndrome and Ellis-van Creveld syndrome) are also associated with congenital hepatic fibrosis. Many cases have concurrent Caroli disease-type changes and are designated as Caroli syndrome.

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Atlas of Liver Pathology by Anthony W. H. Chan, Alberto Quaglia, Beate Haugk, Alastair Burt


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