By R. A. Risdon, D. R. Turner (auth.)
This publication is meant as a realistic bench handbook phological abnormalities in renal illnesses, and for the health facility pathologist who needs to have the place acceptable those were illustrated. entry to an easy informative account of renal even if the most emphasis is at the pathology, pathology, relatively for the translation of the proper scientific features of the stipulations cov percutaneous needle biopsy specimens. I n addition ered are incorporated in acceptance of the truth that we belief it will likely be priceless to physicians operating renal affliction is a space during which correlation of the within the box of renal disorder, for whom the interpre medical and histopathological findings is especially tation of renal biopsy fabric is without delay suitable to big in achieving an educated prognosis. sufferer administration. when a complete insurance extra appro priate to a bigger textual content has no longer been tried, the Acknowledgements textual content has been deliberate to provide an sufficient account of the extra vital non-neoplastic disorder seasoned we want to thank the technical employees of the cesses and their pathological appearances within the Histopathology Laboratories of The London Hos kidney. issues of trouble in interpretation and dif pital clinical collage, The medical institution for unwell Chil ferential prognosis are coated either within the textual content and in dren, nice Ormond road, and Guy's clinic the illustrations.
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275-294. (Oxford: Blackwell). 3. Wilson, C. B. and Dixon, F. J. (1973). Antiglomerular basement membrane antibody-induced glomerulonephritis. Kidnev 1m .. 3, 74--89. 4. Dixon, F. , Feldman, J. D. and Vazquez. J. J. (1961). Experimental glomerulonephritis. The pathogenesis of a laboratory model resembling the spectrum of human glomerulonephritis. J Exp Med, 113,899-920. 5. Soothill. J. F. and Stewart M. V. (1971) The immunopathological significance of the heterogeneity of antibody affinity. Clin Exp.
Lancet, 1,671-675. 8. Spear, G. S. (1974). Pathology of the kidney in cystinosis. In Sommers, S. C. ) Pathology Annual, Vol. 9. pp. 81-92. (New York: Appleton-Century-Croft). 8 G lomeru lonephritis The responses of the specialized capillaries of glomerular tufts to injury are in many ways analogous to those of capillaries elsewhere in the body. The range of resulting morphological changes, and the functional abnormalities produced, provide the structural and clinical expressions ot what is collectively termed glomerulonephritis Such changes may be primary or part of a systemic disease, but the initial damage to the kidney occurs in the glomeruli.
Soothill. J. F. and Stewart M. V. (1971) The immunopathological significance of the heterogeneity of antibody affinity. Clin Exp. , 9, 193-199. 6. Germuth, F. G. and Rodriguez, E. (1973). In ImmunopathologV of the Renal Glomerulus, pp. 81-91. ). Diffuse Endocapillary and Mesangial Prol iferative G lomeru lonephritis Diffuse Endocapillary Proliferative Glomerulonephritis Classically this form of glomerulonephritis follows infections with group A ,B-haemolytic streptococci (usually types 12, 4 or 1 ), and is regarded as a form of soluble complex disease developing during an immune reaction to the infecting organism.
Atlas of Renal Pathology by R. A. Risdon, D. R. Turner (auth.)